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Hyperphosphorylated Tau in AD patients' brains causes configuration changes and loss of tubulin polymerization capacity (Grundke-Iqbal et al., 1986) that result in defective microtubule functioning

Published in-vitro work commonly uses GHK-Cu in the range of 10 nM to 10 M, with 1 M as a frequently reported working concentration

Note: this work used full-length T4, not the TB-500 heptapeptide fragment

6A, Fig S6)

Two-electron reduction of MEN produces a hydroquinone, a pathway that constitutes a detoxification mechanism[51]