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In animal models (and in vitro work), it has been shown to release GH without substantially raising other hormones like ACTH or cortisol, which means less off-target hormonal disturbance

Paracetamol Paracetamol-induced hepatotoxicity represents the result of an imbalance between metabolic detoxication of paracetamol, via conjugation with glucuronic acid and sulphate, and metabolic activation to an unstable toxic metabolite ( N -acetyl- p -benzoquinone imine, NAPQI) via oxidation by cytochrome P450

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It does this by binding to the melanocortin 1 receptor (MC1R)

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The GH signal is clean, the interpulse troughs stay low, and the response-to-administration ratio remains predictable over extended protocol durations