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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

Good news for the needle-nervous: research supports the safety of this approach

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Documented buccal protocols: Buccal strips offer a practical middle ground: bioavailability meaningfully higher than oral capsules, without requiring injection