They are sensed by L cells via FFAR2/GPR43 and FFAR3/GPR41, and activation of these receptors triggers GLP-1 secretion (51)
[28] [29] However, there is currently no robust clinical trial evidence specifically examining GLP-1 medications for hot flash management, and no official link has been established between GLP-1 therapy and vasomotor symptom improvement
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Multiple studies have shown reductions in albuminuria, slower decline in kidney function, and fewer kidney-related complications among patients receiving GLP-1 receptor agonists.[9][10] Then came a study that attracted global attention
Seriously, The GOAT