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Key molecular mechanisms include ROS-mediated MAPK phosphorylation [375], triggering NLRP3 inflammasome assembly [376], oxidative modification of NF-B inhibitor proteins leading to their nuclear translocation [377], and induction of mitochondrial DNA mutations [378]
Notably, the combination of (-)-Vestitol and Salviolone exhibited synergistic effects on reducing the A plaque burden and toxic soluble A levels
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