Although the pathophysiology of endometriosis is not fully explained, retrograde menstruation is the most common theory, and endometriotic cells are known to have a high migratory capacity, be resistant to apoptosis and induce inflammation 3,5,8,9,10,11
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Compared with the control group, the NO-releasing implant increased vascular density in healthy and diabetic pigs by 47.1% and 70.4%, respectively, indicating that NO has a significant vascularization-promoting effect