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Some clinicians also explore it for brain-related goals, but large, definitive trials are still pending

These hormones suppress the activity of these cells and reduce the synthesis of cytokines and mediators involved in the induction of inflammation

In contrast, excessive or unregulated ROS production can compromise mitochondrial membrane potential, trigger cytochrome c (Cyt c) release, and induce oocyte apoptosis through the Bax/Bak-dependent mitochondrial apoptotic pathway, potentially leading to follicular atresia or depletion of the ovarian reserve [34, 35]

Valle A, Saridogan E, Petraglia F, Keckstein J, Polyzos N, Wyns C, et al