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R The crosstalk between NRF2/NQO1, methylation, and glutathione recycling under conditions of high toxic load needs systems-level modeling to understand how these pathways compensate for and compete with each other in real clinical scenarios

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Glutathione plays a critical role in maintaining cellular health by neutralizing harmful free radicals and supporting the body's natural detoxification processes

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Reduced HMG-CoA lyase activity is associated with episodes of hypoglycemia and metabolic acidosis