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In histology, all kidney tissues from the mice treated with colistin showed elevated expression of the AKI markers Ngal and Kim-1, tubule dilation, loss of the brush border membranes in the proximal tubules, and the formation of protein casts throughout the kidney tissue, indicating vast tubular damage (Fig

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Stack It With Pairs Well MOTS-C Both target mitochondrial function - SLU-PP-332 via ERR/PGC-1 pathway, MOTS-C via AMPK