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Such an increase in the mRNA levels of the catalytic subunit of -GCS was paralleled by a greater mucosal synthetic GSH capacity determined from either cysteine or -glutamylcysteine used as the sulfur aminoacid in the presence of glycine and glutamate and ATP as described (Fernndez-Checa and Kaplowitz, 1990), indicating an increase in the activity of -GCS following TNBS+ethanol administration (not shown)

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TB-500 human tolerability studies report mild adverse events at doses up to 1,260 mg [5] [11]

In vitro, ChaC1 induces cell death in KIRC cell lines, signifying its potential as an effective marker for poor prognosis in KIRC (70)