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Cureus, 12(5), e8320

The FAK-paxillin pathway, which regulates cell migration, adhesion, and survival, represents another key mechanism: BPC-157 activates this pathway in fibroblasts and endothelial cells, facilitating the cellular movement required for wound closure and structural tissue regeneration

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A 2021 PubMed review ( Wound Repair and Regeneration ) assessed BPC-157s role in skin wound healing, noting its influence on growth factor pathways in preclinical settings

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