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Uric acid attenuates nitric oxide production by decreasing the interaction between endothelial nitric oxide synthase and calmodulin in human umbilical vein endothelial cells: a mechanism for uric acid-induced cardiovascular disease development
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Milsmann MHW, Schwendener RA, Weder H-G
The post says GLP-1 therapy may not produce significant weight loss in non-overweight patients, but that improved glucose control, cardiovascular protection, and reduced inflammation remain clinically meaningful
Metabolite concentrations were estimated using LCModel [51]
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