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doi:10.1038/ejcn.2011.59 Safer choice

This is likely further enhanced by an increase in GLP-1expressing L-cells in the gut mucosa [9, 16, 17], and gut mucosa hyperplasia that increases intestinal glucose disposal after RYGB [16]

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This section moves beyond a deterministic view of SFN bioavailability to focus on the host factorsspecifically the gut microbiome and host geneticsthat act as key determinants of efficacy