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Genetic rearrangements among the RET tyrosine kinase domain and heterologous genes are the root of RETs carcinogenic potential in PTC

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This altered timing doesn't always cause problems, but it introduces a variable that healthcare providers need to monitor carefully

45 GIP could exert both anti-atherogenic and pro-atherogenic effects, which may depend on the types of endothelia studied (Figure 3)

The mechanisms are well documented

Tech transfer is inherently challenging