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Critical evaluation of receptivity testing illustrates that distinguishing timing misalignment from intrinsic endometrial dysfunction requires multi-layer endotyping beyond transcriptomics alone, while embryo selection benefits from combining metabolic efficiency readouts with morphokinetic parameters and stress response signatures

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Toxicol Sci 65, 30918

Our findings provide a new mechanistic insight into the resistance of IGF-1R inhibition in which MEK1/2 activation stimulates the survival signal (Fig

These approaches address the underlying metabolic dysfunction driving fat accumulation in the liver