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This role is mediated by multiple cytokines, growth factors and death receptor-ligand systems that execute their effect on erythropoiesis partially through apoptosis modulation

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Experimental studies investigate receptor binding affinity, signalling bias, and comparative behaviour relative to other melanocortin analogues

In this context, the antitumor activity reported for incretin-based polyagonists should not be interpreted as evidence for GCGR agonism alone, but rather as an indication that coordinated multi-receptor targeting may induce favorable systemic metabolic remodeling (62,63)

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