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The multi-target therapeutic effects of SCF against MASLD are mediated through diverse molecular mechanisms (see Figure 2), which schematically illustrates the modulation of AMPK-dependent lipid metabolism, PPAR/ signaling pathways, Nrf2/HO-1-mediated antioxidant responses, and PI3K/Akt/mTOR-regulated autophagy, alongside the inhibition of SREBP-1c-driven lipogenesis and endoplasmic reticulum stress

Detoxification Glutathione removes environmental toxinsimportant for mothers exposed to pollution or chemicals

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CD38 Dictates Age-Related NAD Decline and Mitochondrial Dysfunction through an SIRT3-Dependent Mechanism

Once women begin ovarian senescence, estrogen production becomes erratic, antioxidant protection is lost, and oxidative stress is assumed to increase