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Unlike GLP-1 agonists, which primarily act by delaying gastric emptying and enhancing insulin secretion, BRP operates via the following mechanisms: Hypothalamic activation : BRP triggers the expression of the FOS gene, a key marker of neuronal activation associated with appetite suppression, in the hypothalamus

(Monographs of the Mario Negri Institute for Pharmacological Research, Milan)

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In clinical studies, participants have experienced weight loss of 15 to 20 percent over a 72-week period

The review discusses the physiological roles of nitric oxide in mitochondrial energetics and skeletal muscle function