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Generally, two major mechanisms can be envisioned to claim obesogens as prostate carcinogens: i) paracrine signalling by proinflammatory cyto- and chemokines produced by the adipose tissue/PPAT under obesogenic stimulation, creating a chronic inflammatory microenvironment that promotes cancer cell survival, facilitates immune evasion, and supports the spread of malignant cells to distant organs

Here is how that breaks down by purpose

77 ShiA.ZhuC.FuS.WangR.QinG.ChenD.et al (2020)

GHK-Cu delivers the copper required by SOD directly to the cell while simultaneously promoting the expression of the SOD gene

Lancet Diabetes Endocrinol 8:719

KPV is the component that shifts KLOW from purely repair associated discussions toward combined repair plus inflammation modulation study designs