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4, panel B), which remained unchanged throughout the treatment and at 24 weeks was higher than the value recorded in hyperinsulinemic subjects (Fig.4)

It provides a logical explanation for the development of dual targeting drugs such as MM-131 a bispecific anti-MET/EpCAM mAb [108] and also further drug discoveries targeting concurrently all three c-Met, EpCAm and Wnt pathway simultaneously to overcome both innate and acquired resistance to tyrosin kinase inhibitors (TKIs)

This process augments GS phosphorylation, thereby improving hepatic insulin sensitivity (Aurle et al., 2019

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Hartl L, Semmler G, Hofer BS, et al

The majority of patients also tolerate the therapy very well, particularly if in the hands of professional practitioners