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Study on the mechanism of activating SIRT1/Nrf2/p62 pathway to mediate autophagy-dependent ferroptosis to promote healing of diabetic foot ulcers

This activation promotes synaptogenesis, neurogenesis, and neuronal survival by mimicking or potentiating brain-derived neurotrophic factor (BDNF)-like activity, enhancing synaptic connectivity and cognitive plasticity

Both the investigators and the participants

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