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DOI: 10.1128/MCB.22.6.1926-1935.2002

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Competitive inhibitors present an increase in the K m and with V max unaffected

When IGF-1 (or an agonist analog such as IGF-1 LR3) binds the alpha subunits, the receptor undergoes a conformational change that activates the intrinsic tyrosine kinase domains of the beta subunits, triggering autophosphorylation and the recruitment of docking proteins

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