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617 Exposure of astrocytes to -amyloid down-regulates expression of glutamine synthetase, 618 and in animal models, glutamine synthetase expression is reduced in astrocytes surrounding -amyloid plaques

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Autophagy and mitophagy as early barriers to tumor initiation and genomic instability In early tumorigenesis, autophagy functions predominantly as a tumor-suppressive mechanism by removing damaged organelles, misfolded proteins, and oncogenic substrates such as p62, thereby preventing genomic instability and malignant transformation