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The clinical phenotype is variable and has been classified into neonatal onset forms, with the most severely affected patients presenting with congenital anomalies (Type I) or without anomalies (Type II), and mild and/or later onset (Type III) [138]

Particulirement prsent dans les cellules hpatiques, le glutathion prsente galement la capacit de lier, au sein de complexes stables, certaines mtaux et autres xnobiotiques

The primary mechanism documented in published literature is the induction of telomerase catalytic subunit (hTERT) expression in human somatic cells, resulting in measurable telomerase enzyme activity and telomere elongation

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