At subtoxic concentrations, H 2 S has been shown to stimulate ATP production at the level of oxidative phosphorylation, both as a source of electron equivalents for the mETC (via SQR-mediated quinone reduction, Table 1), and through per-sulphidation of ATPase, which maintains the enzyme in its activated state
miR-204 expression is induced by the cellular redox regulator thioredoxin-interacting protein (TXNIP) that then represses MafA, thereby inhibiting insulin production
10.1016/0014-2999(92)90503-v Eur
& Schroeder, M
References Herman CK, Hoschander AS, Wong A
This tripeptide molecule functions as the primary defense mechanism against oxidative stress, free radical damage, and cellular toxin accumulation