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While CKD increases various uremic toxins, clinical evidence has established that indoxyl sulfate serves as an independent, powerful risk factor for cardiovascular mortality and adverse cardiovascular events (67, 68), and its levels also associate with the severity of coronary atherosclerosis (67, 69)

The mean change in HbA1c was not different between the two approaches (WMD 0.08%, 95% CI 1.07 to 1.23)

Indeed, cells exhibit a graded increase in GLP-1 synthesis with increasing metabolic stress (16, 30, 67, 92, 94, 100103)

The ARREST-AF cohort study

It is worth noting that the compounds were tested in vitro using the Caco2 cell line and did not induce cell damage or death, which suggests a favorable preliminary safety profile and supports their potential for future drug development [62]

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