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In oncology, integrate redox proteomics (PTP/cysteine-switch signatures), peroxiredoxin redox states and lipid-peroxide markers to time source-targeted ROS amplification (e.g., radiotherapy/chemotherapy plus ferroptosis inducers) versus node inhibition (NOX, FSP1/CoQ circuits) (Chen et al

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The cardiovascular system is uniquely vulnerable to ferroptotic stress due to its high metabolic demand, elevated oxygen consumption, and dense mitochondrial networks, which together generate substantial reactive oxygen species (ROS)

Current research highlights both the strong safety profile and effectiveness of PRP and exosome therapy

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