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GAPDH acetylation increases GAPDH activity, promotes glycolysis, and enhances memory CD8 + T cell responses
demonstrated that oxidative stress and H 2 O 2 treatment led to a marked increase in senescence-associated -galactosidase activity but only to minimal apoptotic cell death in CSCs, suggesting that ROS triggered the induction of senescence and exhibited therapeutic potential in the eradication of drug-resistant CSCs [8]
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Supported and unsupported rearing were quantified by an observer blind to the treatment conditions using Behavioural Observation Research Interactive Software (BORIS 74 , v.8.7)
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