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The body increases blood flow to the area to repair the micro-trauma, and in doing so, it also flushes out the accumulated metabolic waste and pain-sensitizing chemicals from the original energy crisis

TDP-43 proteinopathy and motor neuron disease in chronic traumatic encephalopathy

Both the parent and redesigned sensors responded as expected, with the new sensors demonstrating intact functionality, as evidenced by reversible changes in fluorescence corresponding to redox potential shifts ( Figure 1b and Figure S2 )

The small sample and historical research methods limit the reliability and generalisability of the findings

Spinal cord injury For many decades, since the 1940s, based purely on correlative observations, reactive astrocytes that surround tissue lesions after traumatic injuries or stroke were regarded as glial scars that were the primary cause for the failure of axon regeneration and functional recovery after spinal cord injury (SCI) and other causes of axotomy in the CNS