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Description

Consequently, intracellular zinc homeostasis alterations regulate downstream molecular objectives, in addition to protein kinase C (PKC), ERK1/2 activation leading to neuronal cell death, cAMP-dependent protein kinase (PKA), Ca/calmodulin-dependent protein kinase II (CaMKII), phosphodiesterases (PDEs), protein tyrosine phosphatases (PTPs), and transcription factors, such as NF-B

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doi: 10.3390/diseases13020059 446 VanVTHLiuZSHsiehYJShiuWCChenBYKuYWet al

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Progress on clinical application of selenium

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doi: 10.1007/s10517-008-0297-x

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