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The study result indicated its most likely because of new protein synthesis and the activation of the mTOR pathway (a driver for increased muscle mass)

Patients show different results when undergoing GLP-1 medication treatments

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By blocking TREK-1, PE-22-28: Increases neuronal excitability : Reduces potassium efflux, depolarizing neurons toward firing threshold Enhances serotonergic transmission : TREK-1 is expressed on serotonergic neurons in the dorsal raphe nucleus

Numerous mechanisms contribute to this action, including the reduction of ROS generation and the encouragement of Nrf2 nuclear translocation, which increases heme oxygenase-1 (HO-1) expression and lowers oxidative stress [175,176,177]