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In conclusion, CIT serves as a metabolically active regulator of NO-dependent signaling, mitochondrial function, and protein metabolism, thereby establishing itself as a significant candidate for nutritional interventions aimed at preserving muscle and vascular integrity during the aging process (Figure 2)

Dovio A, Perazzolo L, Osella G, Ventura M, Termine A, Milano E

People who inject while standing or with a bent, tensed leg consistently report more discomfort

Matsumura, S

Ultrastructurally atrophic hepatocytes have reduced amounts of glycogen and decreased numbers of mitochondria

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