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Results Mitochondrial defects in GNE deficient cells Our previous reports indicated that mutations in GNE disrupted mitochondrial structure and function leading to cell apoptosis 16

A controlled human study did not find a significant effect on stimulated ACTH or cortisol release

Abstract The anti-oxidative enzyme, glutathione peroxidase 4 (GPX4), helps to promote inflammation resolution by eliminating oxidative species produced by the arachidonic acid (AA) metabolic network

This is one of the few convergences seen in the heterogeneous context of autism and provides a good foundation for pharmacological target development