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It is my theory that increasing dermal IGF-1 levels can help to overcome receptor and pathway mutations by increasing the rate of signaling, promoting more consistent keratinocyte differentiation and maturation, while in parallel also stimulating sebocyte proliferation through 5-Reductase and inhibiting inflammation via stimulation of IL-10 production

Upon stimulation, the PI3K produces AKT phosphorylation which has exposed to have a synergistic impact on NF-B signaling

Unlike oral B12, which depends on intrinsic factor and healthy gut absorption, the injection delivers B12 directly into the bloodstream

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doi: 10.1038/srep01408