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Induction of apoptosis by curcumin: mediation by glutathione S-transferase P1-1 inhibition
In the context of the present invention, a base is considered as a substance capable of forming a cation in a solution, particularly in an aqueous and aqueous/alcoholic solution
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By using a reducible disulfide linker, we built in a redox-sensitive release mechanism to facilitate intracellular release of the unmodified NMDA receptor antagonist, enabling the combined cellular actions of GLP-1 receptor agonism and NMDA receptor antagonism
The drug is also released from the microparticles as the polymeric excipient bioerodes
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