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As the Si-based agent was effective in treating UC in the brain and large intestine of mice, it was considered to be capable of suppressing exacerbations and sustaining remission of UC

doi:10.1111/acel.12368 Lu J, Wang Z, Cao J, Chen Y, Dong Y

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During one experiment using a murine model, Pal-GHK was suggested to suppress the activation of Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-B) and p38 mitogen-activated protein kinase (MAPK) signaling pathways, both of which are associated with inflammation