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Metabotropic receptor-dependent long-term depression persists in the absence of protein synthesis in the mouse model of fragile X syndrome

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This might be the indicative proof of the general concept for the recovery from muscle disability (counteracting the stasis of peripheral and central thrombosis, with a direct effect on the maintenance of the thrombocytes function but without affecting coagulation pathways) [5,6,66,71,72,73,74,75,76,77,78,79,80,81,82]