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The FDA-approved drug diflunisal can also inhibit ACMSD, providing a structural base from which derivatives can be designed 237

Previous work has established that any residual OCR detected after oligomycin A treatment is due to non-respiratory oxygen consumption, which is usually ascribed to proton leakage through the mitochondrial inner membrane 79

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In animal studies, TB500 has shown the ability to stimulate the regeneration of damaged cardiac muscle, a phenomenon critical in the recovery from heart-related issues