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In conclusion, BPC-157 promotes the ex vivo outgrowth of tendon fibroblasts from tendon explants, cell survival under stress, and the in vitro migration of tendon fibroblasts, which is likely mediated by the activation of the FAK-paxillin pathway

For NPs with similar chemical compositions, different surface modifications lead to toxic effects in varying degrees (Zhang et al., 2021)

Both compounds interact with fundamental healing processes, but through different pathways that create additive or synergistic effects when combined

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