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Similarly, ATMs in lean subjects are supported by fatty acid oxidation (FAO), glycolysis and glutaminolysis (anti-inflammatory macrophage), while ATMs in obese subjects depend more on glycolysis (pro-inflammatory macrophage) [9]

AOD-9604 failed to demonstrate clinical efficacy and has no current therapeutic application in weight management

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Other treatment options include an IV dose of acetyl-L-carnitine 5 mg/kg bolus loading dose followed by 0.025, 0.1, or 1 mg/kg constant infusion