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& Mohamet, L

doi: 10.1203/00006450-199208000-00018 126 WaltherFJIkegamiMWarburtonDPolkDH

Mct2 overexpression promotes recovery of cognitive function by increasing mitochondrial biogenesis in a rat model of stroke

This results from a tyramine-induced depletion of norepinephrine normally present in the readily releasable vesicle pool, and replacement of vesicular norepinephrine with tyramine (which functions as a false- or pseudo- neurotransmitter), which when released has no effect on postsynaptic adrenergic receptors (Figure 1)

PfK13 mutations have also been shown to be associated with an increased baseline stress response 16, 17, 20

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