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The proposed mechanism involves Epithalon-induced telomerase reactivation in T-cell progenitor populations, extending their replicative lifespan and maintaining thymic output
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NRF2 expression also induces the expression of NQO1, heme-oxygenase 1 (HO1), and FTH1, affecting intracellular iron metabolism, and these processes can be inhibited by p62 activation (57)
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