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Finally, the model predicts that p62 knock-down by siSQSTM1 yields lower base levels of Keap1-GFP in the autophagosome and also lower levels upon DEM treatment relative to the siCtrl condition (Fig 3C

Nonobese diabetic mice express aspects of both type 1 and type 2 diabetes

While omics and systems biology approaches offer valuable molecular insights, linking these findings to clinical outcomes remains a significant challenge

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Smolen GA, Sordella R, Muir B, Mohapatra G, Barmettler A, Archibald H, et al