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GHK-Cu downregulates acute-phase and pro-inflammatory cytokines (TNF-?, IL-6), upregulates anti-oxidant defenses (including superoxide dismutase, Nrf2 and HO-1 pathways), and suppresses fibrotic signaling by inhibiting TGF-?1/Smad2/3 activation

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The body uses B12 slowly over time, with excess amounts stored for future use, allowing the vitamin to remain in the body for weeks or even months

GHK-Cu's clinical dataset is smaller but contains some results worth noting