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10.4239/wjd.v13.i3.129 6 ArasuA.MoranL

In particular, a growing body of in vivo evidence supports the concept that some cancers become functionally dependent on FSP1, creating a therapeutic vulnerability that can be exploited either as monotherapy in specific contexts or as combination therapy with ferroptosis-inducing treatments

Neri S, Swinkels DW, Matlung HL, van Bruggen R

Supports structured energy intake reduction research: By activating satiety circuits and slowing digestion, Cagrilintide reduces caloric intake in controlled experimental settings