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doi: 10.2147/JIR.S456000 209 WirleitnerBNeurauterGSchrcksnadelKFrickBFuchsD
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Timestamps: 0:00 Cold open the zero-studies finding 1:21 Why "overtrained" does four different jobs simultaneously 16:10 The FOR / NFOR / OTS taxonomy 19:43 The supercompensation model borrowed from endurance, never validated for resistance training 32:28 Austin's clinical differential for fatigue and declining performance 36:17 RT evidence what happens when researchers try to induce OTS through lifting 43:19 Austin what actually drives the complaints he sees in practice 47:30 Six theories for what causes overtraining syndrome 1:01:09 The biomarker problem why the T:C ratio and cortisol don't work 1:05:09 What your wearable is actually measuring (and what it isn't) 1:09:28 Austin testosterone levels in trained athletes and when to act 1:13:40 Heart rate variability limitations for strength training 1:15:36 Session RPE the monitoring tool that actually works 1:17:31 How common is overtraining syndrome, really
6.3.2 Insulin modulation HA represents the endocrine hallmark of PCOS, sustained mainly by hyperinsulinemia, OS, and disrupted steroidogenic signaling (96)
