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208,209 Similarly, apoptotic cell death in brain tissues from AD patients and patients with familial AD carrying PA-1 mutations is usually correlated with the intracellular accumulation of A42

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doi: 10.1158/1078-0432.CCR-14-1630 149 WeiXMaoLChiZShengXCuiCKongYet al

It, thus, appears that these interventions do not counteract specific aspects of the pathophysiology of the individual movement disorders, but non-specifically replace and block abnormal activity in the basal ganglia networks that target the relatively intact downstream portions of the motor circuitry [294,295,296]

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