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Kong et al., similarly uncovered synergy between a complex I inhibitor and a mito-antioxidant in cancer cells, suggesting their ability to reduce mitochondria-derived H 2 O 2 levels as a potential mechanism of action (28)

Wang, M., Firrman, J., Zhang, L., Arango-Argoty, G., Tomasula, P., Liu, L., Xiao, W., & Yam, K

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566 In addition to targeting the TFs, PROTACS have also been developed against transcription co-activators with an aim to indirectly regulate TF-associated disease phenotypes