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Epithelial and endothelial damage, in turn, causes translocation of pro-inflammatory mediators and bacterial products, worsening systemic inflammation [23,43]
A., Lyons, M
Although the pathogenic role of oxidative stress in NAFLD pathogenesis is well established (5), there are limited studies available in the literature investigating the potential effect of glutathione supplementation in this condition
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Using three different glioblastoma (GBM) cell lines, namely LN18, A172 and U118, we have recently shown a clear difference between the metabolic profiles of GBM cells, exosomes and their media [156]
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