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We have shown that bTNF- induces a metabolic change in bFLS by significantly increasing glycolysis components and decreasing some components of the TCA cycle, which suggests that the inflammatory effect of bTNF- on bFLS is glucose metabolism-dependent

We next assessed the role of endogenous glutathione in the redox balance of the periplasm

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But their regulatory trajectories differ

These factors lead to lipid peroxidation of the sperm membrane, decreased motility, and increased sperm DNA fragmentation, ultimately compromising fertilization capacity [9, 14]